If One Remedy Fails

practice seems simple to ignorant person.More one sticks to the basic materia medica and organon better the chances of doing good work......the old is gold in Homoeopathy as well as they had guts to accept failures and then they worked out hard to achieve the mission to cure.....with open mind .

You are completely wrong to say those people would not give sensation - all patients give sensation, it is found in the Generals, Modalities, Descriptions of pain etc. Those kind of comments continue to display  ignorance of the method and of Sankaran's work . All his work is based on the work of the old homoeopaths, is more in tune with Boenninghausen and Hahnemann than Kent's method. Whatever reasons he might have for not treating certain patients or going to certain places, this would not be it. And I do not believe the 'old masters' were travelling into Africa either to treat patients. Perhaps we could speculate about their love of comfort, their fear of the unknown or their racial prejudices - all of which would be nothing but speculation.

Well you are wrong, plain and simple. But of course you are entitled to your opinion. I post more cases here on this forum than anyone else does - I am constantly backing up my claims with experience not quotes. You must be thinking of someone else.

And as an aside, I actually have African patients, many people from parts of Africa are being resettled in Brisbane. I have whole families of african people, including children. All display sensation and respond to remedies elicited using the new method. In fact in many ways they can express their problems more easily as they can use gesture to describe their pains and sensations to me.

The practical application of Sankaran method is in the early stage. Homeopath does understand him. The problem is that illiterate people cannot express themselves and you cannot draw information from them unless you are an expert in psychology and have sufficient time.
sajjad.

I have practiced for more than 20 years in rural Bihar, one of the most backward areas of India. Then I have practiced in rural Orissa, another state still living in the abyss of ignorance of basic sanitary practices and poverty.

There were all kinds of cases, even region-specific problems like Kala-jar and Chicken gunea in addition to the myriad of problems known to mankind. Most of them responded beautifully to the methods of Classical Homeopathy.

My teachers have been Hahnemann, Boenninghaeusen, Hering, Gross, Allen, Kent, Dunham, Burnett and Farrington. I never had to go beyond these masters and I am proud to say that I could find enough satisfaction in my 30 years to clinical practice to enter into a very contended retired life.

When I came to Bombay, many of my old friends told me about Sankaran's methods. I read him on the Sensation method and his so-called new methods of Miasmatic prescribing. I was disappointed! This guy, in the name of making Clinical Practice simpler has made all sort of absurd classifications. He calls ringworm and typhoid a miasm! Why? How do they in any way taint the entire system or exhibit any genetic pre-disposition?

The only concept of his that I find a little sensible is trying to find remedies by family characteristics. But, even this is nothing ut old wine in a new bottle! Clarke and Farrington have already led much stress to family classification of remedies and thei familial picturs - a concept Mr. Little has so beautifully explained to the new initiates as the genus Materia Medica.

Parting comments : Homeopathy in itself is a complete systems. the masters have left us a system that will never fail us if applied correctly. And please, let nobody tell me that Lyco is not a tri-miasmatic and other such funny stuff as Sankaran would like us to believe.

Keep smiling

Warm regards
Niel

If one looks at Sankaran's method as a system of case-taking, I have no objection. But to say that so-called deeper layers cannot be explored through classical methods is wrong.

I, in my practice, have often used the Socratic approach of case-taking and that can tell you all about the patient that you may want to know for the purpose of prescription.

Besides, Sankaran's methods are limited not just to proper case-taking. He divides remedies into water-tight miasmatic compartments which is patently wrong.

Keep smiling

Regards
Niel

Originally posted by N Madhavan But to say that so-called deeper layers cannot be explored through classical methods is wrong.

And Niel, I think that reading a book and practicing the method are quite different - practicing it brings results, reading the books is somewhat confusing. It is not surprising you found it difficult to grasp or even believe.  When I first came in contact with the method, through a seminar by Divya Sankaran, I found this style of case taking incomprehensible - it was just too different to how I had been trained. Years later, with much practice, it makes perfect sense and works very efficiently alongside the more traditional case taking techniques.

Those 'taints' (referring to several of the new miasms) do in fact affect every system of the body. The patterns associated with these illnesses (leprosy, malaria, typhoid) can appear in mulitple family members.

Let me share with you some scientific research about Leprosy....

"Two McGill scientists are leading an international research team that announced a breakthrough last week: they have isolated a section of a human chromosome that makes people vulnerable to leprosy.

"Medicine and human genetics professor Erwin Schurr and human genetics professor Tom Hudson were two of the co-authors of a research paper announcing the discovery in the March issue of the journal Nature Genetics. The announcement has already stirred considerable interest, for a problem that Canadians may think is relegated to the past.

"That depends on where you live," said Schurr, a genetics researcher at the McGill University Health Centre. "On a global scale, there are 500,000 to 700,000 new cases each year. These cases are not equally distributed around the world; there are a few high incidence countries that account for the vast majority of cases."

Schurr led a team that went to some of those countries to collect DNA samples from members of affected families. The most afflicted countries are India and Brazil; some African, Asian and South American nations also have new cases every year.

The disease is very rare in Europe and North America and most of the developed world, and the number of cases in afflicted countries has gone down thanks to aggressive multi-drug chemotherapy. Symptoms of leprosy include skin lesions, permanent nerve damage leading to numbness of the feet and hands and, if left untreated, the disease may result in gross disfiguration including loss of finger, toes, feet and hands. The leprosy bacteria are transmitted through direct personal contact.

"Before the introduction of drug therapies 20 years ago, there were about 3 million new cases a year. So the caseload came down, but the question is why hasn't it gone away completely? We believe our discovery is an answer to that."

While the team has not yet identified the actual gene, they have narrowed down its location to a specific area on human chromosome 6.

"The gene predisposes people to becoming infected; it provides a friendly environment for the bacterium (Mycobacterium leprae, the known cause of the disease)."

Tom Hudson, director of the McGill University and Genome Quebec Innovation Centre, says that the gene is almost certainly a mutant version of a healthy gene carried by all humans. He says the significance of the breakthrough can be placed in the context of long-term and ongoing efforts to show a genetic component to infection.

"People at the McGill Centre for Host Resistance — Schurr, (Philippe) Gros, (Emil) Skamene and others — have been working for two decades to show that we get infectious disease not just because of bugs, but also because of genes that make us susceptible. The group has shown in the past that genes played this role in animals, but this is one of the first times that the genetic link has been shown in humans."

The research team for this study used state-of-the-art gene mapping techniques to look for a common thread between affected families."

So, you can see that in fact the susceptibility to Leprosy does in fact become passed down. Homoeopathy realised this before science did (again!)

And in regards to Malaria, let me share another interesting piece of science....

"An inherited childrens' blood disorder called alpha thalassemia can protect children against malaria, according to a new study.

"It is really remarkable and so simple. Children with alpha thalassemia have adapted to the loss of red blood cells associated with malarial disease by making more of these cells with less hemoglobin," says Dr. Day. "So, these children do better because they end up with more hemoglobin overall when they have a malaria attack compared to normal children," says Dr. Day.

Malaria afflicts hundreds of millions of people, causing up to 2 million deaths every year in Africa and Asia. Many of its victims are young children. In regions of the world where malaria is endemic, mutations have arisen in human populations that allow people to survive. Sickle cell trait, for example, protects against malaria.

So here we have an inherited genetic trait that has arisen as a result of Malaria in previous generations - which clearly fulfills your requirements for classifcation as a miasm.

Now typhoid is a little harder to clearly see this way as Leprosy and Malaria is. But I found this interesting article...

'A key to survival of Salmonella typhi is its ability to lie dormant in carriers, who show no symptoms but remain able to infect others,' says Kathryn Holt, a PhD student at the Wellcome Trust Sanger Institute and first author on the study. 'Our new tools will assist us in tracing the source of typhoid outbreaks, potentially even to infected carriers, allowing those individuals to be treated to prevent further spread of the disease.

'Using the genomic biology of this study, we can now type typhi, identify the strain that is causing infection, identify carriers and direct vaccination programmes most efficiently. It is a remarkable step forward.'

The study is a collaboration between researchers at the Wellcome Trust Sanger Institute, University College, Cork, Institut Pasteur in Paris and Oxford University Clinical Research Unit, Ho Chi Minh City, Vietnam. The team studied 19 isolates of typhi from ten countries, using new sequencing methods that meant they could capture the rare signals of genetic variation in this stubborn genome. They produced more than 1.7 billion letters of genetic sequence and found evidence of fewer than 2000 mutation events, suggesting very little evolution since the emergence of typhi at least 15,000 years ago.

Their analysis shows that the typhi genome is decaying - as it becomes more closely allied to us, its human host, it is losing genes that are superfluous to life in the human body. More importantly, genes that contain instructions for the proteins on the surface of the bacterium - those most often attacked by our immune system defences - vary much less than do the equivalent genes in most other bacteria, suggesting that typhi has a strategy to circumvent the selective pressures of our immune system."

So Typhoid has adapted to the human host exclusively, and it can sit dormant in a person, making them a carrier and allowing them to infect others. That is very similar to other illnesses that are associated with miasms (like Tuberculosis).

And this same article, suggests a similar reaction to Typhoid, as others have had to Malaria ...

"The gene mutation that causes cystic fibrosis also appears to protect against infection with typhoid fever bacteria, a study supported by the National Institute of Allergy and Infectious Diseases (NIAID) has found. The finding could explain why an estimated 12 million people in the United States carry the gene for such a highly fatal childhood disease. Researchers led by Gerald Pier, Ph.D., of Brigham and Women's Hospital and Harvard Medical School in Boston, report the finding in the May 7, 1998, issue of the journal Nature."

So another genetic mutation, an inherited illness, that has a relationship with Typhoid. While not as clear as the Malaria link, it is certainly interesting.